New research from UW’s School of Medicine and Public Health, funded in part by the Wisconsin Partnership Program, found that a new therapeutic approach for heart failure could help restore cardiac function by regenerating heart muscle. In a study recently published in the journal Circulation, the UW team described how the function of heart muscle in a mouse was improved by temporarily blocking a key metabolic enzyme after a heart attack. “Our research shows that it may be possible to improve the function of the heart muscle after infarction, which is good news for people who have systolic heart failure,” says UW’s Ahmed Mahmoud, PhD, assistant professor of cell and regenerative biology.
